Lastly, the molecular mechanisms of BST in preventing cell proliferation via pro-apoptotic, anti-metastatic, anti-invasive, and chemo-sensitizing actions that include factors such as p53 protein, B cell lymphoma-2 (Bcl-2), ROS, cell cycle blockade, interleukins, vascular endothelial growth factor (VEGF), mitogen activated protein kinase (MAPK), and PCNA pathways with applications to prostate disorders, but tumor progression, in general, have been recently reviewed [134]. This evidence concerns the gene VEGFA and prostate disorder.