Regardless of gender, the other potential regulators of the plasma FSAP levels were the following: higher body mass index (BMI) [57], hypertriglyceridemia, high fibrinogen levels [34,57], elevated levels of plasminogen activator inhibitor type 1 (PAI-1), thrombin activatable fibrinolysis inhibitor (TAFI), and TAFI activation peptide [57]. This evidence concerns the gene CPB2 and hypertriglyceridemia.