CPT1A and obstructive sleep apnea syndrome: Despite striking differences in glucose metabolism indices, time spent in hypoxia, and the number of apneic/hypopneic episodes, no differences in skeletal muscle palmitate oxidation capacity were observed between the patients with and without severe OSA, which was further corroborated by the unchanged protein and gene expressions of the essential proteins involved in FFA cellular transport and oxidation, i.e., CD36, FAPT4, and CPT1.