The polymorphism of the FCGR2A, FCGR2C and FCGR3A genes found in patients with sarcoidosis may cause dysfunction of the Fcγ receptors encoded by them, and thus explain the previously observed increased immunocomplexemia with a simultaneous increase in the percentage of FcγR monocytes in the same patients [16,112,113]. This evidence concerns the gene FCGR2A and sarcoidosis.