In another molecular study, Dubaniewicz et al. [8] assessed the polymorphism of the FCGR genes encoding the receptor for the Fc fragment of immunoglobulin G IIa, IIb, IIc, IIIa and IIIb (FcγRIIa, FcγRIIb, FcγRIIc, FcγRIIIa and FcγRIIIb, respectively) plays a role in enhancing the circulation of immune complexes with the presence of M heat shock proteins in the case of tuberculosis in patients with sarcoidosis. Here, FCGR3A is linked to tuberculosis.