OLR1 and neoplasm: Notably, in CaP, we have previously shown that LOX-1 activation by oxLDLs stimulates tumor angiogenesis [31], epithelial-mesenchymal transition, and tumorigenic potential, being determinant for tumor growth [32], which could explain why obese patients with CaP progress to more malignant stages in a shorter period of time compared to patients with average weight [32].