CXCL8 and endometriosis: The release of cysteine, a precursor of reduced glutathione, from NAC, allows the molecule to perform an indirect antioxidant action, with the removal of ROS leading to the inhibition of proinflammatory cytokines (IL-6, IL-8, TNF-alpha), VEGF, and metalloproteinases, whose concentration is increased in the peritoneum of patients with endometriosis [15,22].