BCL2L1 and autoimmune thrombocytopenic purpura: Increased let-7b-5p in B cells was elevated in ITP, which enhances the expression of surface BAFF Receptor (BAFFR) and phosphorylation of NF-κB2 p100, which in turn promotes increased levels of anti-apoptotic factor, Bcl-xL for B cell survival [69].