VIM and breast carcinoma: These were achieved by inducing the expression of mesenchymal markers, vimentin (Vim), fibroblast growth factor binding protein 1 (FgfBp1), snail family transcriptional blocker-1 (Snail), and β-linked protein; this was also accomplished by upregulating the expression of E-calmodulin, i.e., inhibiting EMT to suppress breast cancer metastasis.