Similarly, it has been observed a significant association between the distribution of volumes of [11C]-PIB and [18F]-FEPPA in the most recent study by Ghadery et al. [18] The correlation between TSPO overexpression by microglial cells and other pathological features of PD and other neurodegenerative disorders can, eventually, provide an additional tool in the future for the use of radiotracers targeting TSPO, as well as in the study of PD pathogenesis and, eventually, in the therapeutic approach to PD patients. This evidence concerns the gene TSPO and Parkinson disease.