STING1 and Sepsis: The fact that COX2, a recognized marker of ferroptosis, was found elevated in the heart of a murine model of sepsis [53,216,217] and the evidence that mitochondrial changes induced by LPS were compatible with mitochondrial features of myocardial ferroptosis [29,214], suggest that ferroptosis may be intimately correlated with the development of septic cardiomyopathy caused by LPS or STING.