Through (3-(4,5-Dimethylthiazol-2-yl)-2,5-Diphenyltetrazolium Bromide (MTT) analysis, the researchers also found that CRC proliferation and colony formation was noticeably enhanced after TMEM100 silencing but significantly weakened after TMEM100 overexpression [29]. The gene discussed is TMEM100; the disease is colorectal carcinoma.