AR and posterior cortical atrophy: This was studied by selecting a collection of PCa biopsies with Gleason Score—GS3 (low) to GS5 (high) via IHC staining and using the pYAP1-Y407 antiserum in comparison to a panYAP1 serum, which revealed that the distribution of pYAP1-Y407 was predominantly nuclear, suggesting that it is a “active” form of the protein capable of AR or TEAD transactivation.