Interestingly, by multi-platform omics analyses, Radovich and colleagues [27] revealed tumoral overexpression of genes encoding muscle and muscle-like autoantigens, including NEFM (mimicking AChR-α and TTN) and RYR3 (mimicking RYR1), in MG compared to non-MG thymomas, also showing molecular differences across WHO types that could be of pathogenic relevance for thymoma-associated MG. The gene discussed is NEFM; the disease is myasthenia gravis.