In contrast, no clear treatment or method has been found for AD, and recent studies have shown that several mechanisms are involved in the deposition of β- amyloid (Aβ) plaques, Aβ aggregation, neuronal death, cholinergic nervous system diseases, the formation of NFTs with abnormal phosphorylated tau, oxidative stress, neuroinflammation, and genetic factors [6,7]. This evidence concerns the gene MAPT and Alzheimer disease.