Finally, a magnetic bead-based multiplex assay, performed on SNs from KIN I-derived HF primary cultures treated or not with the FGFR2 kinase inhibitor SU5402, indicated that the secretion of the tumor-promoting factor IL-6 is enhanced when FGFR2c is upregulated and activated, and we speculated that an FGFR2c-dependent increase of autophagy could contribute to this phenomenon. The gene discussed is IL6; the disease is neoplasm.