The infection is characterized by high serum levels of IL-1β, IL-2, IL-7, IL-8, IL-9, IL-10, IL-17, G-CSF, GM-CSF, IFN-γ, TNFα, IP10, MCP1, MIP1A, and MIP1B, but, in particular, it influences a sustained increase in IL-6 and IL-1β [21,22,23], which may result in the death of a patient. This evidence concerns the gene IL1B and infection.