KLF15 and heart failure: Regarding the key molecules for decidualization in the autophagy pathway, Oestreich et al. reported that FIP200, whose systemic deletion causes embryonic lethality due to heart failure and liver degeneration [47], was responsible for progesterone-dependent inhibition of epithelial proliferation in the murine endometrium via Klf15 (KLF transcription factor 15) [48].