In addition, we also detected the expression levels of the proteins involved in EMT, a key factor contributing to tumor migration and invasion [29], and the results showed that the expression of epithelial cell marker E-Cadherin was up-regulated, while mesenchymal markers Vimentin and Snail were down-regulated in MCF-7 and MDA-MB-231 cells after exposure to MA as compared with the CT group, indicating that MA inhibits the metastasis of breast cancer cells, and the same results were also obtained by scratch wound assay and transwell assays. The gene discussed is VIM; the disease is neoplasm.