Relevant exposures include bovine spongiform encephalopathy prions in the diet, and use of blood and blood products for variant CJD (vCJD)5; cadaver-sourced human growth hormone,6 neurosurgery, and lyophilized dura mater in iatrogenic CJD (iCJD),7 and at mortuary feasts in the Eastern Highlands Province of Papua New Guinea in kuru.8 This evidence concerns the gene GH1 and variant Creutzfeldt-Jakob disease.