Other potential drugs that promote endogenous hematoma absorption after ICH have also been extensively studied, including wogonin (69), vitamin D (70), and ISO-α-acids (IAAs) (49), which can upregulate surface CD36 of phagocytes via the PPAR-γ pathway, promote the polarization of M2 type microglia, and enhance the phagocytosis of M/MΦ to promote endogenous hematoma absorption after ICH. Here, CD36 is linked to hematoma.