AKT1 and nasopharyngeal carcinoma: Expression levels of miR-106a-5p in sEVs are significantly increased in the last cycle of cisplatin-based chemotherapy, and miR-106a-5p is enriched in cisplatin-resistant cell-derived EVs and promotes cisplatin resistance in NPC cells in vivo by regulating the ARNT2/AKT axis (Li J. et al., 2021).