During our experiments, overexpression of NMT1 promoted GC cell viability, migration, and invasion while NMT1 knockdown repressed these cell behaviors, consistent with the functions of NOX4, MFN2, as well as Rac1 on GC [27–29], which determines the oncogenic role of NMT1 in GC. This evidence concerns the gene NMT1 and gastric cancer.