DMD and Duchenne muscular dystrophy: DMD phenotype in mdx mice was partially corrected by in utero intramuscular injections of high‐capacity AdV129 and intraperitoneal injections of AAV8130 at E16, both vectors expressing the minidystrophin gene, restoring dystrophin‐associated glycoprotein complex in muscle fibers with improved histology and functionality, albeit with low expression levels following AdV‐IUGT due to anti‐AdV antibodies.