Knowledge gained on the EBV‐associated antigen profile in the autopsied brains of MS patients49, 54 and our understanding of the immune evasion mechanisms of the latency II proteins provides the basis for the hypothesis that the inefficient control of EBV‐infected B cells expressing EBNA1 and LMP1 &2 contributes to the pathogenesis of MS.65, 66. Here, PDLIM7 is linked to myeloid sarcoma.