AKT2 and urinary bladder carcinoma: miR-148a also repressed proliferation of bladder cancer cells by establishing a positive feedback loop between ERBB3/AKT2/c-myc signaling and DNMT1 [22] or by regulating expression levels of DNMT1 and undifferentiated embryonic cell transcription factor 1 (UTF1) in cervical cancer cells [23].