When the common oral microorganism Fusobacterium nucleatum strain F7‐1 and altered Schaedler's flora (ASF) colonize the intestinal tract of germ‐free mice, SCFAs produced by Fusobacterium nucleatum increase the risk of intestinal cancer by binding to FFAR2 and promote the expression of IL‐17, favoring the occurrence of an inflammatory environment prior to tumor formation.[51]. The gene discussed is FFAR2; the disease is neoplasm.