BNIP3 and digestive system infectious disorder: Together, these data indicate that host autophagy is hijacked by SARS-CoV-2 to establish intestinal infection and underscore the therapeutic potential of targeting the endosomal/autophagy entry route, in particular via BNIP3-based strategies directed at autophagosome-lysosome fusion, to suppress intestinal SARS-CoV-2 entry and replication as well as limit SARS-CoV-2-mediated intestinal barrier damage.