SMOC1 and COVID-19: The fact that OAS genes were highly expressed in both SARS-CoV-2-infected cardiomyocytes and human failing hearts provides us a reason to believe that some similar signaling molecules and/or pathways may mediate the developments of COVID-19 and HF, or in other words, there may be some similar or common molecular mechanisms in the two diseases, and OAS gene family may be the common genetic factors.