Molecule Activity Prediction (MAP) analysis predicted inhibition of TGFB1 cytokine as a central node, suggesting interference with mitogenic pathways that involve TGFB receptor signaling in cancer, i.e., cell proliferation and epithelial-to-mesenchymal transition (EMT) [9], a core process of fibrosis and wound healing [34]. This evidence concerns the gene TGFB1 and cancer.