Two way ANOVA showed that there was no significant interaction between AKI and diets for Bax levels in the kidney [F (4,50) = 2.451, P = 0.0580], but the main effects of AKI and diets were significant for Bax levels [F (1,50) = 291.5, P < 0.001] and [F (4,50) = 42.36, P < 0.001], respectively.The post hoc Tukey test revealed that in animals without AKI, ER, and TR diets decreased renal Bax compared to the CTL group (P < 0.05), while HF increased this index compared to the ER, TR (P < 0.001), and IF (P < 0.05) diets. This evidence concerns the gene BAX and acute kidney injury.