TJP1 and hepatocellular carcinoma: Highly intrahepatic metastatic hepatocellular carcinoma (HCC)‐cell‐derived exosomal miRNAs, including miR‐638, miR‐663a, miR‐3648, and miR‐4258, attenuated endothelial junction integrity and promoted vascular permeability by decreasing the endothelial expression of vascular endothelial‐cadherin (VE‐cadherin) and zonula occludens‐1 (ZO‐1), thus initiating liver PMN formation.78