Overexpression of RAB22A was linked to processes such as cell adhesion via the plasma membrane (35), nonsense-mediated decay of nuclear-transcribed mRNA (36), axon guidance (37), oxidative phosphorylation (38), FCGR activation (39), and eukaryotic translation elongation (40) in a GSEA of HCC. This evidence concerns the gene RAB22A and hepatocellular carcinoma.