In recent years, antibodies targeted proteins of the nodal/paranodal regions, including neurofascin 155 (NF155), neurofascin 186 (NF186), contactin‐1 (CNTN1) and contactin‐associated protein 1 (Caspr1), have been reported in the cases of CIDP, which were also known as autoimmune nodopathies and showed specific clinical characteristics compared with the classical CIDP.9, 10, 11, 12. This evidence concerns the gene CNTNAP1 and chronic inflammatory demyelinating polyradiculoneuropathy.