And that proved the regular intake of UDCA 6 months after surgery reduced incidence of cholelithiasis after OAGB.[35] UDCA, a hydrophilic secondary bile acid, increases biliary acids that help to form biliary micelles and vesicles, which decreases bile lithogenicity, biliary secretion of cholesterol and mucin, cholesterol saturation, and intestinal absorption.[36] Our study corroborates the findings of previous studies that indicated the effectiveness of UDCA in lowering the incidence of cholelithiasis post-BS. Here, MUC5AC is linked to cholelithiasis.