Exogenously added CTH attenuates H2O2‐induced and doxorubicin‐induced apoptosis and maintains mitochondrial stability by increasing intra‐cytoplasmic calcium concentration, restoring the number of mitochondrial cristae, and increasing mitochondrial reserve and exerting anti‐apoptotic effects in BC cells46 (Figure 1). The gene discussed is CTH; the disease is breast cancer.