Regarding its role in terms of mediating IVIg efficacy, it has to be noted that although CD32a is known to be suppressed by IVIg, our data and a previous study indicate that this mechanism of action does not seem to play a major role in the pathogenesis in CIDP (Quast et al. 2015). Here, FCGR2A is linked to chronic inflammatory demyelinating polyradiculoneuropathy.