To verify whether ERK inhibition affected Rab32-Drp1 interactions, we performed the Co-IP after Rab32-HA-transfected GBM cells treated with SCH772984 and the data showed that inhibition of ERK1/2 reduced the binding of Rab32 and Drp1, suggesting that the interaction of Rab32 with Drp1 is partly dependent on the ERK1/2 activation (Fig. S6D). This evidence concerns the gene MAPK3 and glioblastoma.