HIF1A and myocardial infarction: As MI results from acute and prolonged deficits in oxygen supply, which trigger a series of severe biochemical and metabolic disturbances in the cardiomyocytes [11], we attempted to determine the potential role of one of hypoxia-induced lncRNA, hypoxia-inducible factor 1A antisense RNA 2 (HIF1A-AS2) as a potential biomarker for AMI.