In addition, Li and Zhang (26) found that miR-708-5p influenced M. tuberculosis viability and the human macrophage inflammatory response during infection by targeting TLR4 and miR-708-5p mimics reduced proinflammatory factors including IFN-γ, interleukin-6 (IL-6), IL-1β, and TNF-α. The gene discussed is TLR4; the disease is infection.