Pathogenic variants in ATP2C1 cause Hailey‐Hailey disease, another autosomal dominant skin disorder.[44] Impairment of ATP2C1 leads to a dysfunction in the Golgi‐associated human secretory pathway Ca2+/Mn2+ ATPase (hSPCA1) that results in recurrent blisters and erosions in intertriginous sites.[45] Vulvar lesions are also possible.[44, 46, 47] This causes pain, vulvodynia, pruritus, and the development of chronic malodorous abnormal growths at risk of infection with Staphylococcus aureus and Candida albicans. This evidence concerns the gene ATP2C1 and infection.