This suggests that hyperactivity, working memory deficits, and increased sociability might be related to the developmental roles of FGFR2 in astrocytes in the neonatal period, whereas the modest decrease in anxiety-like behavior might reflect a continuous, ongoing role of FGFR2 in the functioning of astroglial cells as we previously demonstrated for short term memory [8]. This evidence concerns the gene FGFR2 and Anxiety.