Because hyperactivity, working memory deficits, and increased sociability were present here when FGFR2 was lacking from the neonatal period onward but not when the loss was induced in adulthood, these results support a new line of thinking, implicating fibroblast growth factor signaling in the early postnatal brain [59] and further suggest that these processes may affect the risk for behavioral disorders [38]. The gene discussed is FGFR2; the disease is Atypical behavior.