In this context, Kelly and co-authors demonstrated that the commensal Bacteroidetes Thetaiotaomicron blocks the dysfunctional acute inflammatory response to infection by pathogenic Salmonella enterica by inducing binding of PPARγ to NF-κB RelA subunit and their joint nuclear export and cytosolic localization, resulting in the inhibition of the transcription of pro-inflammatory cytokine IL-8 [31]. This evidence concerns the gene NFKB1 and infection.