SLC1A2 and Menkes disease: Sipe and collaborators used a transgenic mouse line with constitutive heterozygous expression of the GLT1 (SLC1A2, GLT1-HET) and observed that MD in heterozygous mice lead to an aberrant OD plasticity in the ipsilateral non-deprived eye, characterized by an excessive depression of the responses (Figure 1M) [34].