In another study carried out in CS cells and in vivo in mice, overexpression of breast cancer anti-estrogen resistance 4 (BCAR4), a long noncoding RNA (lncRNA) that participates in the formation of multiple cancers [84], resulted in hyperacetylation of histone H3 in the mammalian target of rapamycin (MTOR) promoter. This evidence concerns the gene MTOR and breast carcinoma.