The pathogenic and risk factor genetic variants in the SOX2, FBXO38, and RNASEL genes are the possible candidates for the modifiers of the disease manifestation, since the mentioned SNVs were found in the GBA-PD patient’s exome and were absent in the GBA-carrier (FBXO38 and RNASEL), or were present in the homozygous state of GBA-PD and are heterozygous in the GBA-carrier (SOX2) (Table S3). This evidence concerns the gene RNASEL and Parkinson disease.