This result contrasts with a previous study, in which miR-93 showed to be underexpressed in the ectopic endometrium of patients with endometriosis compared to the peritoneal tissue of patients without the disease, causing increased expression of MMP3 and VEGFA, thus stimulating the proliferation, migration, and invasive capacity of endometrial stromal cells [45]. Here, VEGFA is linked to endometriosis.