COL17A1 and idiopathic pulmonary fibrosis: Konigsberg and colleagues combined four omics datasets (protein-coding RNA, protein, DNA methylation, and noncoding RNA) from IPF and healthy lung tissues to construct a multi-omics network, and their results confirmed previously validated molecules (i.e., MMP7, AGER, COL17A1, TNXB, LAMC3, and RARA-AS1) and pathways already known to be dysregulated in IPF disease [10].