DGAT1 and Ureteral obstruction: Supporting this, our group has recently reported that SFN alleviated FA metabolism dysfunction in the unilateral ureteral obstruction (UUO) model by downregulating cluster of differentiation 36 (CD36) levels and the FA synthesis proteins, such as FA synthase (FAS), sterol regulatory-element binding protein 1 (SREBP1) and diacylglycerol O-acyltransferase 1 (DGAT1), as well as triglyceride levels in the renal tissue [56].