Specifically, TNFSF14 and GAD1 are highly expressed in HCC [54]; STARD1 promotes the progression of non-alcoholic steatohepatitis to HCC via bile acids [55]; DHRS4-AS1 ameliorates HCC by suppressing proliferation and promoting apoptosis via the miR-522-3p/SOCS5 axis [56]. This evidence concerns the gene GAD1 and metabolic dysfunction-associated steatohepatitis.