NEAT1 has been reported to participate in multiple diabetic metabolic syndrome, which accelerates the occurrence and development of diabetic nephropathy by sponging miR-23c.[24] Others have claimed that lncRNA NEAT1 regulated diabetic retinal epithelial-mesenchymal transition via regulating miR-204/SOX4 axis.[25] Our research demonstrated that the expression level of NEAT1 was significantly higher in the model group, which might be related to m6A modification. Here, SOX4 is linked to diabetic kidney disease.